Treatments May Fuel Cancer's Spread?

Albert Einstein, the Great Man of Science wrote:(cell death) and motility. At the early stage of
"It is theory that decides what we can observe."carcinogenesis (cancer process) TGF-beta is a
If our mind is set (biased by the theory that wepotent inhibitor of cell proliferation but at later
have learnt in school) then "that is all" that we canstages of carcinogenesis the levels of TGF-beta
see - we can't see anything beyond what weincrease with tumour progression. It is not known
have learnt or been taught! It needs anhow TGF-beta switches it role from being a
open-minded person to "see" what others do nottumour suppressor to being a promoter during
want to see or fail to see.the course of cancer progression.
For twenty-six years, I devoted two lectures inMany disease processes are associated with loss
my physiology class to teaching universityof normal TGF-beta function, such as cancer,
students understand the Scientific Method. Theatherosclerosis, autoimmune and inflammatory
first step for any scientific discovery isdiseases. Excessive TGF-beta production have
OBSERVATION of a phenomenon. If you cannotbeen implicated in the formation of scar tissues or
or don't want to "see" how can you everfibrosis (in lung and liver), development of
discover anything new?pulmonary edema (fluid in lung),
After more than a decade working with cancerimmunosuppression and successful parasite
patients there is one phenomenon that I hadinfection.
observed: " breast cancer patients tend to sufferTGF-beta has been said to cause chronic
metastasis (spread) to their lungs, bones, etc.pulmonary (lung) fibrosis in rats and mice exposed
pretty soon after undergoing medical treatments."to chemo-drugs, bleomycin or cyclophosphamide,
Of course, some doctors may scream at such aand in the development of hepatic (liver) fibrosis in
suggestion! I wonder if this "hunch" is an illusion?rats exposed to radiation.
The answer came on 7 April 2007, in the form ofTGF-beta has been shown to promote breast
a small, insignificant column in The Star. It had thiscancer metastasis by acting directly on the
heading: "Study: Treatment may fuel cancer'stumour cells. TGF-beta is also shown to enhance
spread." The study reported in the Journal ofparathyroid-hormone-related protein (PTHrP) and
Clinical Investigation by Dr. Carlos Arteaga andsubsequently resulted in bone destruction in breast
colleagues at Vanderbilt University, USA, showedcancer patients. Advanced breast cancer patients
that treating cancer with surgery, chemotherapyhave been shown to have increased plasma
or radiation may sometimes cause tumours toTGF-beta levels after chemotherapy and
spread. In their work they used doxorubicin (aTGF-beta is positively associated with disease
common chemo-drug used for breast cancer) orprogression.
radiation and found that these treatments raisedDr. Carlos Arteaga and colleagues showed that
levels of TGF-beta, which in turn helped breastwhen mice infected with human breast cancer
cancer tumours to spread to the lung. Thewere treated with radiation or doxorubicin, they
researchers wrote: "The repopulation andhad higher levels of TGF-beta in their blood. They
progression of tumours after anti-cancer therapyalso had more tiny tumour cells in their blood and
(such as radiotherapy, chemotherapy andthese cells spread to the lungs. When the mice
surgery) is a well-recognised phenomenon."were treated with antibody that suppresses
How many of us (doctors and patients alike)TGF-beta, the spread stopped. The spreading
know this? If doctors know, do they tell theirprocess did not occur at all in mice bred to lack
patients? If patients know, what is theirTFG-beta.
response?Comments: What are the implications of the
After reading this report, I spent a whole morningabove? When you are asked to do chemo or
surfing the web to learn more about thisradiation, ask first what is the main objective of
phenomenon. This is what I learnt. The key to oursuch a treatment - to cure? to prevent cancer
understanding is a biological protein calledfrom spreading? or what. This study showed that
Transforming Growth Factor beta (TGF-beta).treatments like chemotherapy or radiation cause
TGF-beta exists in at least five (iso)forms, knownthe spread of cancer. This report is a direct
as TGF beta 1, beta 2, beta 3, beta 4 and beta 5.opposite of what you are often told about the
The roles of TGF-beta in cancer are verypurpose of chemotherapy or radiotherapy - i.e., to
complex and often confusing indeed. TGF-betakill the cancer cells or stop them from spreading!
controls cell proliferation, differentiation, apoptosis